Refractory Angina

CONTENTS Number 16, 2002

	EDITORIAL Editorial G. Jackson
	BASIC ARTICLE Cardiac ischemic pain F. Crea, A. Gaspardone
	MAIN CLINICAL ARTICLE Diagnosis, incidence, epidemiology, and treatment of refractory angina M. Chester
	IMAGING PET imaging in refractory angina P.G. Camici
	NEW THERAPEUTIC APPROACHES TMR, PMR, and SCS D.C. McNab, P.M. Schofield
	FOCUS ON VASTAREL Cardioprotection during myocardial revascularization: benefit of a metabolic intervention I. Holban
	CASE REPORT Refractory angina G. Jackson
	REFRESHER CORNER The physiology of pain perception in angina pectoris R.D. Foreman
	FEATURED RESEARCH Abstracts and commentaries
	ALL ARTICLES AS PDF FILE For easy printing and true layout; to be viewed with free Adobe® Acrobat® Reader®

The figure shows PET images of rubidium-82 (Rb) and F18 fluorodeoxyglucose (FDG) uptake in the left ventricle of a patient with stable angina. In each image the left ventricle free wall is in the 6 to 10 o'clock position, the anterior wall and septurn are in the 1 to 3 o'clock position and the remaining open area is the plane of the mitral valve. The perfusion scan recorded at peak exercise (top right) shows a severely reduced Rb uptake in the anterior left ventricle wall compared to the scan recorded at rest (top left). The FDG scan recorded following an injection of FDG in the recovery phase (bottom right), when Rb had normalized (bottom left), shows a higher (1.90 times) tracer concentration in the previously ischemic region as compared to the nonischernic tissue (free wall). From: Camici PG, Araujo L, Spinks T et al. Increased uptake of F18 flurodeoxyglucose in postischemicmyocardium of patients with exercise-induced angina. Circulation 1986;74:81–88.